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Oscience. Trends Neurosci. 1999;22(4):167-173. PubMed Silverman ES, Collins T. Pathways of Egr-1-mediated gene transcription in vascular biology. Am J Pathol. 1999;154(3):665-70. doi:10.1016/S00029440(10)65312-6. PubMed PMID: 10079243; PMCID: PMC1866415. Thiel G, Cibelli G. Regulation of life and death by the zinc finger transcription factor Egr-1. J Cell Physiol. 2002;193(3):287-292. doi:10.1002
Defect model in rabbits. The major merit of this study was that, this implant has been tested in vivo so that with a welldesigned pilot and experimental study we were able to explain the mechanism of action of this implant on tendon healing. In addition, we followed the immune activity of the body in response to the implanted collagen prosthesis from the early stages to four months after injury b
Described experiments. SM performed real-time PCR analysis of EGR2, CD163, and HLA-DRA in human mammary explant tissue and performed data analysis. SS participated in the study design, edited the manuscript, and gave final approval of the version to be published. All authors read and approved the final manuscript. Competing interests The authors do not have any financial or personal relationships
Riments performed in duplicate and normalized to the amplification of ACTB mRNA. Bars represent mean ?SEM of the fold change with respect to untreated. *p
Poprotein; SFRP1: Secreted frizzled-related protein-1; TAM: Tumor Associated Macrophage; TGF-: Transforming growth factor- Acknowledgements Not applicable.5.6.7. Funding The design of this study, collection, analysis, interpretation, and writing of the manuscript were made possible because of the funding support by the Rays of Hope Foundation. Availability of data and materials The raw files are
Described experiments. SM performed real-time PCR analysis of EGR2, CD163, and HLA-DRA in human mammary explant tissue and performed data analysis. SS participated in the study design, edited the manuscript, and gave final approval of the version to be published. All authors read and approved the final manuscript. Competing interests The authors do not have any financial or personal relationships
Mass consists of tumor-associated macrophages (TAMs) and poor prognosis is associated with elevated levels of TAMs [44]. Stimuli in the tumor environment polarize TAMs towards a protumor M2 rather than an anti-tumor M1 phenotype [45]. TGF- promotes tumor progression by recruiting TAMs to compete with dendritic cells by suppressing their antigen-presentation [34]. Zhang et al. provide evidence tha
Oscience. Trends Neurosci. 1999;22(4):167-173. PubMed Silverman ES, Collins T. Pathways of Egr-1-mediated gene transcription in vascular biology. Am J Pathol. 1999;154(3):665-70. doi:10.1016/S00029440(10)65312-6. PubMed PMID: 10079243; PMCID: PMC1866415. Thiel G, Cibelli G. Regulation of life and death by the zinc finger transcription factor Egr-1. J Cell Physiol. 2002;193(3):287-292. doi:10.1002
Ey MJ, Uren A, Schaudies RP, Popescu NC, Rudikoff S, Aaronson SA, Varmus HE, Rubin JS. Purification and molecular cloning of a secreted, Frizzled-related antagonist of Wnt action. Proc Natl Acad Sci U S A. 1997;94(13):6770-6775. PubMed 2. Bafico A, Gazit A, Pramila T, Finch PW, Yaniv A, Aaronson SA. Interaction of frizzled related protein (FRP) with Wnt ligands and the frizzled receptor suggests
Ey MJ, Uren A, Schaudies RP, Popescu NC, Rudikoff S, Aaronson SA, Varmus HE, Rubin JS. Purification and molecular cloning of a secreted, Frizzled-related antagonist of Wnt action. Proc Natl Acad Sci U S A. 1997;94(13):6770-6775. PubMed 2. Bafico A, Gazit A, Pramila T, Finch PW, Yaniv A, Aaronson SA. Interaction of frizzled related protein (FRP) with Wnt ligands and the frizzled receptor suggests
Rolled them in order to debride and proliferate throughout the collagen implant in the defect area.Meimandi-Parizi et al. Journal of Biomedical Science 2013, 20:28 http://www.jbiomedsci.com/content/20/1/Page 16 ofFigure 7 Histopathological findings (Part 2) 120 days after injury (Results section: Histopathological findings). There is fatty degeneration in the injured area of the ICTs. The density
Described experiments. SM performed real-time PCR analysis of EGR2, CD163, and HLA-DRA in human mammary explant tissue and performed data analysis. SS participated in the study design, edited the manuscript, and gave final approval of the version to be published. All authors read and approved the final manuscript. Competing interests The authors do not have any financial or personal relationships
Oscience. Trends Neurosci. 1999;22(4):167-173. PubMed Silverman ES, Collins T. Pathways of Egr-1-mediated gene transcription in vascular biology. Am J Pathol. 1999;154(3):665-70. doi:10.1016/S00029440(10)65312-6. PubMed PMID: 10079243; PMCID: PMC1866415. Thiel G, Cibelli G. Regulation of life and death by the zinc finger transcription factor Egr-1. J Cell Physiol. 2002;193(3):287-292. doi:10.1002
Rolled them in order to debride and proliferate throughout the collagen implant in the defect area.Meimandi-Parizi et al. Journal of Biomedical Science 2013, 20:28 http://www.jbiomedsci.com/content/20/1/Page 16 ofFigure 7 Histopathological findings (Part 2) 120 days after injury (Results section: Histopathological findings). There is fatty degeneration in the injured area of the ICTs. The density


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