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Described experiments. SM performed real-time PCR analysis of EGR2, CD163, and HLA-DRA in human mammary explant tissue and performed data analysis. SS participated in the study design, edited the manuscript, and gave final approval of the version to be published. All authors read and approved the final manuscript. Competing interests The authors do not have any financial or personal relationships
Described experiments. SM performed real-time PCR analysis of EGR2, CD163, and HLA-DRA in human mammary explant tissue and performed data analysis. SS participated in the study design, edited the manuscript, and gave final approval of the version to be published. All authors read and approved the final manuscript. Competing interests The authors do not have any financial or personal relationships
Defect model in rabbits. The major merit of this study was that, this implant has been tested in vivo so that with a welldesigned pilot and experimental study we were able to explain the mechanism of action of this implant on tendon healing. In addition, we followed the immune activity of the body in response to the implanted collagen prosthesis from the early stages to four months after injury b
Defect model in rabbits. The major merit of this study was that, this implant has been tested in vivo so that with a welldesigned pilot and experimental study we were able to explain the mechanism of action of this implant on tendon healing. In addition, we followed the immune activity of the body in response to the implanted collagen prosthesis from the early stages to four months after injury b
Described experiments. SM performed real-time PCR analysis of EGR2, CD163, and HLA-DRA in human mammary explant tissue and performed data analysis. SS participated in the study design, edited the manuscript, and gave final approval of the version to be published. All authors read and approved the final manuscript. Competing interests The authors do not have any financial or personal relationships
Ey MJ, Uren A, Schaudies RP, Popescu NC, Rudikoff S, Aaronson SA, Varmus HE, Rubin JS. Purification and molecular cloning of a secreted, Frizzled-related antagonist of Wnt action. Proc Natl Acad Sci U S A. 1997;94(13):6770-6775. PubMed 2. Bafico A, Gazit A, Pramila T, Finch PW, Yaniv A, Aaronson SA. Interaction of frizzled related protein (FRP) with Wnt ligands and the frizzled receptor suggests
Ey MJ, Uren A, Schaudies RP, Popescu NC, Rudikoff S, Aaronson SA, Varmus HE, Rubin JS. Purification and molecular cloning of a secreted, Frizzled-related antagonist of Wnt action. Proc Natl Acad Sci U S A. 1997;94(13):6770-6775. PubMed 2. Bafico A, Gazit A, Pramila T, Finch PW, Yaniv A, Aaronson SA. Interaction of frizzled related protein (FRP) with Wnt ligands and the frizzled receptor suggests
Ey MJ, Uren A, Schaudies RP, Popescu NC, Rudikoff S, Aaronson SA, Varmus HE, Rubin JS. Purification and molecular cloning of a secreted, Frizzled-related antagonist of Wnt action. Proc Natl Acad Sci U S A. 1997;94(13):6770-6775. PubMed 2. Bafico A, Gazit A, Pramila T, Finch PW, Yaniv A, Aaronson SA. Interaction of frizzled related protein (FRP) with Wnt ligands and the frizzled receptor suggests
Expression. The results shown represent experiments performed in duplicate and normalized to the amplification of ACTB mRNA. Bars represent mean ?SEM of the fold change with respect to vector transfected control cells. *p
Expression. The results shown represent experiments performed in duplicate and normalized to the amplification of ACTB mRNA. Bars represent mean ?SEM of the fold change with respect to vector transfected control cells. *p
E harvests for real-time PCR analysis of Egr2. All real-time PCR results are from two separate experiments performed in triplicate and results were normalized to amplification of ACTB mRNA. Bars represent mean ?SEM and are expressed as fold change with respect DMSO treated cells. *p
Described experiments. SM performed real-time PCR analysis of EGR2, CD163, and HLA-DRA in human mammary explant tissue and performed data analysis. SS participated in the study design, edited the manuscript, and gave final approval of the version to be published. All authors read and approved the final manuscript. Competing interests The authors do not have any financial or personal relationships
Riments performed in duplicate and normalized to the amplification of ACTB mRNA. Bars represent mean ?SEM of the fold change with respect to untreated. *p
Oscience. Trends Neurosci. 1999;22(4):167-173. PubMed Silverman ES, Collins T. Pathways of Egr-1-mediated gene transcription in vascular biology. Am J Pathol. 1999;154(3):665-70. doi:10.1016/S00029440(10)65312-6. PubMed PMID: 10079243; PMCID: PMC1866415. Thiel G, Cibelli G. Regulation of life and death by the zinc finger transcription factor Egr-1. J Cell Physiol. 2002;193(3):287-292. doi:10.1002


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